Why angina pain radiate

People with angina pectoris or sometimes referred to as stable angina have episodes of chest pain. The discomfort that are usually predictable and manageable. Normally this type of chest discomfort is relieved with rest, nitroglycerin or both.

Nitroglycerin relaxes the coronary arteries and other blood vessels, reducing the amount of blood that returns to the heart and easing the heart's workload.

By relaxing the coronary arteries, it increases the heart's blood supply. If you experience chest discomfort, be sure and visit your doctor for a complete evaluation and, possibly, tests. If you have stable angina and start getting chest pain more easily and more often, see your doctor immediately as you may be experiencing early signs of unstable angina. Written by American Heart Association editorial staff and reviewed by science and medicine advisers. See our editorial policies and staff.

Heart Attack. About Heart Attacks. Warning Signs of a Heart Attack. Angina Chest Pain. It can be triggered by physical exertion or stress and is thought to be due to spasm of the artery. Microvascular angina: Pain caused by abnormal function of the tiny microscopic arteries of the heart. Medications to reduce the progression of coronary artery disease.

Medications to reduce symptoms of angina. Changes in lifestyle. Improved control of risk factors such as diabetes, high blood pressure or tobacco use. Cardiac rehabilitation. Seek immediate medical attention if your symptoms are new or alarming.

Make an Appointment To schedule an appointment to discuss your need for angina treatment, call us at or visit our Make a Cardiovascular Appointment page, where you may fill out a Patient Appointment Request Form and view other information about scheduling a cardiovascular appointment. Vagal cardiac afferent fibers likely mediate atypical anginal pain and contribute to cardiac ischemia without accompanying pain via relays through the nucleus of the solitary tract and the C1-C2 spinal segments.

The psychological state of an individual can modulate cardiac nociception via pathways involving the amygdala. Descending pathways originating from nucleus raphe magnus and the pons also can modulate cardiac nociception. Sensory input from other visceral organs can mimic cardiac pain due to convergence of this input with cardiac input onto spinothalamic tract neurons.